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Erythrocytes affect hemostasis and thrombosis by altering blood viscosity, blood flow dynamics, secreting RBC-derived microvesicles, interactions with platelets or endothelial cells, fibrinogen-induced RBC aggregation, activation of blood coagulation factors, direct modulation of thrombin and fibrin formation, or formation of polyhedral-like erythrocytes in thrombi [47,48,49]

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133 The complex degradation pathway of Amadori products forms small electrophilic molecules (e.g., glyoxal and methylglyoxal) which can further spontaneously react with protein nucleophiles and form Advanced Glycation End products (AGEs), which have been implicated in numerous chronic pathologies